+肥胖相关内膜不孕新机制!安医大一附院魏兆莲/张东/何晔团队Autophagy(IF 18.6)重磅发文
+IF 24.9|南方医院方媛研究团队发现SPHK1调控结直肠癌肝转移免疫微环境,为免疫治疗提供新靶点
+肠癌患者福音!山东第一医科大学附属中心医院赵蔚冲/孙美丽团队发现KCTD9水平可预知免疫治疗成败,更能提升PD-1效果!
+香港城市大学黄聿团队MedComm发文:空间蛋白组学揭示糖尿病血管病变的区域秘密
+IF 33.9 ! 肖斌/王永权/李薇团队揭示CircABCA1通过重编程胆固醇代谢促进ccRCC的机制
+肾脏细胞自救密码被破译!西农路浩团队发现内质网应激通过UPR通路“遥控”细胞自噬
+华中科技大学同济医学院附属同济医院高红宇/颜奇研究团队揭秘铁死亡与肾间质纤维化的关键联系
+Cell Reports:西湖大学宋春青团队发现SETDB1通过CSF3促进肝脏再生新机制,为肝病治疗带来希望
+IF 13.7 | 赵冰、焦石团队揭示Hippo-YAP信号在肝脏发育和肿瘤发生中的关键作用
皮肤方面:
1.
International Journal of Biological Sciences. (IF=4.858). Wu, et.al. (2019). Metformin Promotes the Survival of Random-Pattern Skin Flaps by Inducing Autophagy via the AMPK-mTOR-TFEB signaling pathway.[温州医科大学附属第二医院 AAV9-U6-shRNA (Prkaa1)-CMV-EGFP 随意皮瓣]
肾脏方面:
EBIOMEDICINE. (IF=5.736). Liu, et.al. (2019). H19 promote calcium oxalate nephrocalcinosis-induced renal tubular epithelial cell injury via a ceRNA pathway.[华中科技大学同济医学院附属同济医院 AAV9-H19 肾钙质沉着症]
2.
Science Translational Medicine. (IF=16.304). Wang, et.al. (2018). Long noncoding RNA lnc-TSI inhibits renal fibrogenesis by negatively regulating the TGF-β/Smad3 pathway.[南方医科大学 AAV9-MIR-GFP-lnc-TSI 肾纤维化]
肺脏方面:
Cell Death & Disease. (IF=6.304). Zhang, et.al. (2019). circHIPK3 regulates lung fibroblast-to-myofibroblast transition by functioning as a competing endogenous RNA.[上海市第一人民医院 AAV6-circHIPK3 shRNA & scrambled shRNA 肺纤维化]
Oncotarget. Tian, et.al. (2017). Sirtuin 6 inhibits epithelial to mesenchymal transition during idiopathic pulmonary fibrosis via inactivating TGF-β1/Smad3 signaling.[华中科技大学同济医学院公共卫生学院 AAV-Sirt6 特发性肺纤维化]
体外实验:
Retrovirology. (IF=4.183). Wang, et.al. (2017). Genome modifcation of CXCR4 by Staphylococcus aureus Cas9 renders cells resistance to HIV-1 infection.[武汉大学基础医学院 AAV-SaCas9-CXCR4-gRNA HIV基因治疗]
巨噬细胞方面:
EBioMedicine. (IF=5.736). Lian, et.al. (2019). Macrophage metabolic reprogramming aggravates aortic dissection through the HIF1α-ADAM17 pathway. [北京大学 AAV2-ADAM17-GFP & AAV2-GFP 主动脉夹层AD]
Circulation Research. (IF=14.467). Fu, et.al. (2016). Shift of Macrophage Phenotype Due to Cartilage Oligomeric Matrix Protein Defciency Drives Atherosclerotic Calcifcation. [北京大学 AAV2-integrin β3 & AAV2-β-tail domain & AAV2-GFP 动脉粥样硬化AS]
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